plko 1 slc16a9 shrna plasmid (Genechem)
86
Structured Review
Genechem
plko 1 slc16a9 shrna plasmid
Plko 1 Slc16a9 Shrna Plasmid, supplied by Genechem, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/plko+1+slc16a9+shrna+plasmid/1+interference+nlrp3+nlrp3+plasmid+plko+sh+shrna/pm42271166-59-29-40
Average 86 stars, based on 1 article reviews
Plko 1 Slc16a9 Shrna Plasmid, supplied by Genechem, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/plko+1+slc16a9+shrna+plasmid/1+interference+nlrp3+nlrp3+plasmid+plko+sh+shrna/pm42271166-59-29-40
Average 86 stars, based on 1 article reviews
plko 1 slc16a9 shrna plasmid - by Bioz Stars,
2026-09
86/100 stars
Images
Related Articles
Transfection:Article Title: SLC16A9-Mediated Carnitine Uptake Enhances Radiotherapy Resistance in Colorectal Cancer via Lipid Metabolic Reprogramming. Article Snippet: Background: Radiation resistance severely impairs the therapeutic efficacy of radiotherapy in colorectal cancer (CRC).. Lipid metabolic reprogramming, particularly the activation of fatty acid oxidation (FAO), has been well recognized to be closely implicated in radioresistance, whereas its underlying regulatory mechanism remains poorly elucidated.. This study aimed to explore the functional role of the transporter SLC16A9 in CRC radioresistance and its correlation with carnitine metabolism as well as the FAO signaling pathway. Over Expression:Article Title: SLC16A9-Mediated Carnitine Uptake Enhances Radiotherapy Resistance in Colorectal Cancer via Lipid Metabolic Reprogramming. Article Snippet: Background: Radiation resistance severely impairs the therapeutic efficacy of radiotherapy in colorectal cancer (CRC).. Lipid metabolic reprogramming, particularly the activation of fatty acid oxidation (FAO), has been well recognized to be closely implicated in radioresistance, whereas its underlying regulatory mechanism remains poorly elucidated.. This study aimed to explore the functional role of the transporter SLC16A9 in CRC radioresistance and its correlation with carnitine metabolism as well as the FAO signaling pathway. Plasmid Preparation:Article Title: SLC16A9-Mediated Carnitine Uptake Enhances Radiotherapy Resistance in Colorectal Cancer via Lipid Metabolic Reprogramming. Article Snippet: Background: Radiation resistance severely impairs the therapeutic efficacy of radiotherapy in colorectal cancer (CRC).. Lipid metabolic reprogramming, particularly the activation of fatty acid oxidation (FAO), has been well recognized to be closely implicated in radioresistance, whereas its underlying regulatory mechanism remains poorly elucidated.. This study aimed to explore the functional role of the transporter SLC16A9 in CRC radioresistance and its correlation with carnitine metabolism as well as the FAO signaling pathway. shRNA:Article Title: SLC16A9-Mediated Carnitine Uptake Enhances Radiotherapy Resistance in Colorectal Cancer via Lipid Metabolic Reprogramming. Article Snippet: Background: Radiation resistance severely impairs the therapeutic efficacy of radiotherapy in colorectal cancer (CRC).. Lipid metabolic reprogramming, particularly the activation of fatty acid oxidation (FAO), has been well recognized to be closely implicated in radioresistance, whereas its underlying regulatory mechanism remains poorly elucidated.. This study aimed to explore the functional role of the transporter SLC16A9 in CRC radioresistance and its correlation with carnitine metabolism as well as the FAO signaling pathway. Synthesized:Article Title: SLC16A9-Mediated Carnitine Uptake Enhances Radiotherapy Resistance in Colorectal Cancer via Lipid Metabolic Reprogramming. Article Snippet: Background: Radiation resistance severely impairs the therapeutic efficacy of radiotherapy in colorectal cancer (CRC).. Lipid metabolic reprogramming, particularly the activation of fatty acid oxidation (FAO), has been well recognized to be closely implicated in radioresistance, whereas its underlying regulatory mechanism remains poorly elucidated.. This study aimed to explore the functional role of the transporter SLC16A9 in CRC radioresistance and its correlation with carnitine metabolism as well as the FAO signaling pathway. |